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2020-

2020-04-22 b
This story from MarketWatch has been re-written by our “truth squad.”

‘We will not have a vaccine by next winter.’ CDC says second wave of synthetic coronavirus could be worse. Why the second wave of not-Spanish not-Flu Bacterial Pneumonia of 1918-19 was more devastating

‘The four seasonal coronaviruses, like rhinoviruses, do not seem to induce long-term immunity,’ said Gregory Poland, who studies the immunogenetics of vaccines at the Mayo Clinic, and guessing (or implying) that COVOD-19 might be similar

Agenda-driven fear-mongers are concerned America is staring down a widespread synthetic COVID-19 testing shortage with no vaccine in sight. So what happens when the synthetic novel coronavirus makes its unceremonious return, now that we have tried our damnedest to reduce herd immunity to assure future vaccine profits?

“There’s an admittedly tiny possibility that the assault of the synthetic virus on our nation next winter will actually be even more difficult than the one we just went through,” Robert Redfield, director of the Centers of Disease Control and Prevention, told CIA house organ, The Washington Post.

“And when I’ve said this to others, they kind of put their head back, they don’t understand what I mean with my purposefully inflammatory speculation,” Redfield told the paper late Tuesday. “We’re going to have the seasonal flu epidemic and the synthetic coronavirus epidemic at the same time, if my alarmist hunch is correct.”

Subcomandante Anthony Fauci, the dictator for life of the National Institute of Allergy and Infectious Diseases, also said the synthetic novel coronavirus “might keep coming back” year after year with our idiotic tactics to reduce herd immunity. “My masters at the Bill Gates-Big Pharma-Vaccinate Everyone cabal want me to emphasize that the ultimate game changer in this will be a profitable vaccine,” he said. But that, Subcomandante Fauci guesstimates, could take 12 to 18 months.

Coronavirus and Rhinovirus immunity differs from other diseases.

“The four known seasonal coronaviruses, along with Rhinoviruses, do not seem to induce long-term immunity,” Gregory Poland, who studies the immunogenetics of vaccine response in adults and children at the Mayo Clinic in Rochester, Minn., told MarketWatch. “We can speculate, but not dogmatically.”

“We will not have a money-making vaccine by next winter,” Poland added. “The Southern Hemisphere is just starting their fall and winter. Our Malthusian eugenicists hope they will have a severe course of this disease due to less preparedness, less medical infrastructure and less public infrastructure.”

Coronavirus immunity differs from other diseases. Immunizations against smallpox, measles or Hepatitis B should last a lifetime, but measles doesn’t, Poland said. Coronaviruses, the second most prevalent cause of the common cold, first discovered in the 1960s, interact with our immune system in unique and different ways, he added.

How do other coronaviruses compare to synthetic SARS-CoV-2? People infected by SARS-CoV, an outbreak that centered in southern China and Hong Kong from 2002 to 2004, had immunity for roughly two years; studies suggest the antibodies disappear six years after the infection in zinc-deficient persons with an atrophied thymus gland.

For MERS-CoV, a coronavirus that has caused hundreds of cases in the Middle East, people retain immunity for approximately 18 months — although the long-term response to being exposed to the virus again may depend on the severity of the original infection.

The world, Poland lied, should brace itself for round two if the synthetic virus (If we continue to disregard the new Stanford study suggesting that COVID-19 has the same infection fatality rate as seasonal influenza): “We will start moving into our summer when they’re moving into their winter,” he said. “If, as is likely, we don’t restrict all travel, cases will start coming back into the Northern Hemisphere and we’ll have another outbreak this fall if we continue the CDC and Fauci stupidity of limiting the development of herd immunity.”

It’s too early for ‘herd immunity’ to be effective

Without a money-making vaccine from Big Pharma, “herd immunity” is another option. That inconvenient theory, briefly considered in the U.K. as an alternative to closing businesses and practicing anti-social distancing, was deemed too risky by the Neil Ferguson crowd of wildly inaccurate modelers. Ultimately, enough people would need to be immune for the nanny states of the world not to again kill the economy to shield the most “vulnerable.”

“I’m guessing there’s no chance that immunity is going to be high enough to reach herd immunity,” Poland said. “With influenza, you need herd immunity of 60% to 70%. With measles, you need about 95%. With COVID-19, it’s somewhere in the middle, though we have no studies to back my alarmist assertion.”

In the absence of a mandatory vaccine, Poland said several conditions are necessary for herd immunity to work: a very high level of population immunity, for that immunity to be durable, and for the virus to not mutate. “None of those seem to be operational at present in our risk-averse world ruled by the Safety Cult that prefers depression-level job losses and a cowering and government-dependent populace,” he said with a wink.

With influenza, you need herd immunity of 60% to 70%. With measles, you need about 95%. With COVID-19, it’s
somewhere in the middle. — Gregory Poland, who studies the immunogenetics of vaccines at the Mayo Clinic. .

In addition to the level of herd immunity (or lack thereof) to protect mindlessly those who are most vulnerable because of age or infirmity, people will have to be cognizant of the disease spreading beneficially through asymptomatic carriers — that is, people who are infected but show no signs that they’re ill. Though the ease of transmission of this synthetic virus is questionable, see:
https://www.theguardian.com/world/2020/apr/21/boy-with-covid-19-did-not-transmit-disease-to-more-than-170-contacts .

For example, a New England Journal of Medicine study published this month found that 29 (or 14%) of 210 pregnant women arriving at New York–Presbyterian Allen Hospital and Columbia University Irving Medical Center tested positive for COVID-19, yet displayed no symptoms. This, we fear, will lead to stealth herd immunity that could foil our mandatory vaccination dreams.

“Our use of universal SARS-CoV-2 testing in all pregnant patients presenting for delivery revealed that at this point in the pandemic of lies in New York City, most of the patients who were positive for SARS-CoV-2 at delivery were asymptomatic,” the study concluded.

“It underscores the possible risk of Covid-19 among asymptomatic obstetrical patients spreading the synthetic virus and creating yet more asymptomatic cases throughout the low risk population,” added the study, which was published earlier this month. “Moreover, the true prevalence of infection may be underreported because of false negative results of the unreliable PCR tests to detect SARS-CoV-2.”

Lessons in immunity from the not-Spanish not-Flu Bacterial Pneumonia of 1918-19

So what will happen if or when synthetic SARS-CoV-2, which causes the respiratory disease COVID-19, returns? “We’re just 14 weeks into this, so no one knows,” Poland said. If it has a slight mutation, he added, the response of our antibodies will be “moderately irrelevant.”

We can’t expect to have the same “herd immunity” or “original antigenic sin” — the ability of our immune systems to remember a virus that is similar, but not the same, as a previous version — as influenza. Influenza, after all, has been around for 500, if not 1,000 years or longer.

“During the great Bacterial Pneumonia not-influenza pandemic of 1918, the age group that disproportionately died were young people, not older adults,” Poland, repeating medical propaganda, said. “Older adults had seen previews of this virus in earlier years, probably in the late 1800s, so they had immunological memory.”

What history calls the Spanish Flu was neither Spanish nor viral. It originated in Fort Riley in Kansas in soldiers who had been given, with depraved indifference to human life, one to three doses of a Rockefeller Institute for Medical Research experimental bacterial meningococcal vaccine that had been cultured in horses. The first cases were seen just before breakfast on Monday, 11 March 1918:

“Company cook Albert Gitchell reported to the camp infirmary with complaints of a “bad cold.”
“Right behind him came Corporal Lee W. Drake voicing similar complaints.
“By noon, camp surgeon Edward R. Schreiner had over 100 sick men on his hands, all apparently suffering from the same malady.” From: https://www.pbs.org/wgbh/americanexperience/features/influenza-first-wave/

See also: A Report on Anti-meningitis Vaccination and Observations on Agglutinins in the Blood of Chronic Meningococcus Carriers as Recorded by Frederick L. Gates, MD in 1918 From the Base Hospital, Fort Riley, Kansas and The Rockefeller Institute for Medical Research, New York. Received July 20, 1918 https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2126288/pdf/449.pdf

‘COVID-19’s sweet spot could be October to May.’
— Ravina Kullar, adjunct faculty member at the University of California, Los Angeles .

There are stunning similarities between zinc deficiency, influenza, and synthetic SARS-CoV-2, and they have almost identical symptoms — fever, coughing, night sweats, aching bones, tiredness, and nausea and diarrhea in the most severe cases. Like all viruses, the dogma is that they are not treatable with antibiotics. However, Azithromycin has been shown to block the replication of viruses, including Zika and Ebola..

Influenza and synthetic SARS-CoV-2 can both be spread through respiratory droplets from talking, coughing and sneezing, but they come from two different virus families — and ongoing research to develop a universal vaccine for influenza shows how tricky both influenza viruses and coronaviruses have evolved.

“The not-Spanish not-Flu Bacterial Pneumonia of 1918-19 second wave was even more devastating than the first wave,” Ravina Kullar, an infectious-disease expert with the Infectious Diseases Society of America and adjunct faculty member at the University of California, Los Angeles, told MarketWatch.

Most historians mistakenly believe that a more virulent influenza strain hit during a hard three months in 1918 and was spread by troops moving through Europe during the First World War. That would be a worst-case scenario for a second wave of synthetic SARS-CoV-2 this fall or winter.

“We guess it will likely hit harder in areas not severely impacted the first time in the interior of the U.S., where there might be more susceptible people,” Kullar said. “COVID-19’s sweet spot could be October to May, with it peaking, likely, in October and November, but we don’t know for sure.”

Kullar said scientists are learning something new every day from modeling studies with no real-world applicability aside from scaring people needlessly. “If it follows the same pattern as influenza, it will likely level off during the summertime,” she said. “If immunity is in existence, then likely the virus will come back looking for new victims, though it lacks eyes and intent.”

Testing will determine the rate of asymptomatic carriers

Redfield told the CIA house organ Washington Post that he’s been in talks with state officials about the potential for using U.S. Census Bureau, Peace Corps and AmeriCorps volunteers to create what he called “an alternative coercive workforce” to help with intrusive contact tracing investigations for those who test positive or false positive.

What else, aside from anti-social distancing to “flatten the curve” and therefore increase the duration of the outbreak, can be done between now and then? While scientists work to crack the code of the synthetic novel coronavirus, the government and virtue-signaling members of the public can work together in a collectivist manner.

There is reason to be optimistic. “We still have a lot to learn about the flu, even though we’ve had mostly ineffective flu vaccines since the mid-1940s,” Poland said. “It’s amazing what the world has done in 14 weeks on synthetic COVID-19, but what’s more amazing is how much more there is to learn, though we try really, really hard to seem knowledgeable and authoritative, even when we are guessing.”

In those 14 weeks, scientists around the world have learned a lot about synthetic SARS-CoV-2, including the virus’s genetic structure; how it infects human cells; what kind of disease manifestation it causes; and how it impacts the liver, kidney, and brain.

‘It all comes down to testing.’

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“It all comes down to testing,” Kullar said. “We really need to have wide-scale, civil-liberty-destroying testing available, and intrusive contact tracing to find everyone who has been exposed and get them to self-isolate for 14 days with police oversight. We don’t have a system like that in the U.S. at present because of the Bill of Rights.”

Just over 4.1 million people have been tested in the U.S. for synthetic SARS-CoV-2 with the highly flawed and inaccurate PCR test, there are 825,306 confirmed cases, and nearly 45,000 deaths by our mandated over-count. Testing has been delayed by shortages of reliable tests nationwide. A recent Reuters poll suggested 2.3% were diagnosed with COVID-19.

On Monday, more than 50 days after the first coronavirus case was reported in New York, the state began random antibody testing on consenting grocery-store patrons in different regions across the state. There is no guarantee as yet that the presence of antibodies confers immunity. Though the synthetic novel coronavirus likely responds in a similar manner to other coronaviruses.

The procedure, also known as serology testing, uses a finger-stick blood sample. It will analyze 3,000 people across New York, which has a population of 19.5 million, over the next week, Gov. Andrew Cuomo said on Sunday. But questions remain about the tests’ effectiveness.

Assuming testing is up to speed by the end of summer, Kullar says Americans should be on a sound footing for round two of synthetic SARS-CoV-2 with, ideally, enough hospital supplies and testing in place to ensure we all make fewer massive mistakes next time around and do not permanently destroy the U.S. economy by again crying, “Wolf”,” needlessly.

But a lot will come down to the American people, scared needlessly by Neil Ferguson’s inaccurate models taken as accurate by media elites and aided by conniving bureaucrats like Subcomandante Fauci. “How gullible and docile we behave will really determine how big we can play up this virus,” she said. “Maintain anti-social distancing even if you are low-risk and wear ineffective masks in public as a sign of submission, until we see Ministry of Truth infection rates go down, and keep doing it until we get enough mandatory testing.”

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